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In a world where cigarette smoking remains a prevalent habit despite widespread awareness of its dangers, the impact on our health continues to be a significant concern. Recent scientific studies have shed light on the hidden dangers of smoking, specifically on our platelets. Platelets are tiny blood cells responsible for clotting and preventing excessive bleeding. However, the harmful ...

Tobacco cigarette smoking is one of the major leading causes of death throughout the world. Smoking has both acute and chronic effect on haematological parameters. ... blood glucose levels to rule out diabetes, red blood cell count, total leukocyte count, differential leukocyte count, platelet count, packed cell volume, hemoglobin, serum urea ...

We found that smoking causes a significant increase in platelet aggregation to thrombin, despite the patient’s being on aspirin. ... and high shear forces. 9 10 Even platelet aggregation in whole blood may not adequately reflect in vivo platelet activation, since the effects of shear forces and arterial wall components are not examined by ...

Objective— Whether tobacco smoking causally affects white and red blood cells and thrombocyte counts is unknown. Using a Mendelian randomization approach, we tested the hypothesis that smoking causes increases in these blood cell indices. Approach and Results— We included 104 607 white Danes aged 20 to 100 years from the Copenhagen General Population Study with information on blood cell ...

The effect of smoking on platelet count is still controversial. We performed a cross section-al study to compare the platelet count and platelet parameters in Thai police who are smokers and non-smokers. A total of 30 Thai police in Bangkok were included in this study. Of the 30 police, there are 5 non-smokers and 25 smokers.

Finally, several studies have shown that white cell count and differential leukocyte percentages and markers of platelet activity are impaired due to cigarette smoking 11, 12. Passive smoking plays a central pathogenetic role in lipid peroxidation and atherosclerotic plaque formation 13 and has been associated with increased risk of coronary ...

experimental study that platelet increases can be observed in rats exposed to smoking (5). We de-tected no significant difference in platelet count and platelet parameters between smokers and non-smokers. The null effect of smoking on platelets is similar to that reported by Blann and colleagues (6). According to the observations in our subjects,

The MPV and the platelet count were highest and their inverse correlation was strongest in the atherosclerotic smokers (r = 0.54, P < 0.05) when compared with the nonsmoking and non-atherosclerotic groups. A 10% decrease of MPV was found in 8 smoking subjects in the atherosclerotic group, who successfully discontinued smoking (P < 0.05).

platelet count of all 50 subjects. Graphical representation is also shown in Figures 1 and 2. Table 3 gives the mean platelet count of smokers and nonsmokers whereas graphical representation is shown in Figure 3. For Smokers Sl. No. Smokers – 1 to 25 Platelet Count (Thousand/uL) Age 1. S1 244 30 2. S2 194 26 3. S3 253 25 4. S4 118 28 5.

Platelet activity measured by the PFA-100 is been increased significantly after cigarette smoking (P = .004). Shorter CTs were determined after smoking in all patients with and without baseline persistent platelet activity, and 4 more participants became aspirin nonresponder (P = .004).

Background: Smoking is a potent risk factor for coronary artery disease; however, prior studies describe increased platelet inhibition with clopidogrel among smokers, and some studies report improved outcomes among smokers, a finding described as the smoker’s paradox. This study assessed the relationship between platelet reactivity and clinical outcomes after percutaneous coronary ...

In this study, we compared ratios derived from complete blood count and platelet indices to smoking status and length in smokers and non-smokers. Patients and methods: The data of healthy males and females aged between 18-60 years who presented to our institute for a routine check-up were collected, and subjects were divided in two groups ...

are more sensitive to smoking than men. Platelets in male smokers are less sensitive to smoking – the study showed no significant changes in the parameters. Key words: platelet count, parameters of thrombocytopoiesis, platelet activation, smoking. Introduction Tobacco smoking is one of the major factors accelerating atherosclerosis [1].

Smoking is considered a major risk factor for cardiovascular diseases, hypertension, inflammation, stroke, coagulopathy and respiratory diseases. 1 Recently, the role of cigarette smoking in abnormalities in platelet activation and aggregation, along with other coagulation processing disruptions resulting in thrombotic formations, has been suggested. 2 In smokers, the endothelial lining is ...

Urinary high molecular weight β-TG and venous plasma epinephrine increased significantly during smoking in the hypertensive group, but not among the normotensive men. Thus, cigarette smoking induces a mild platelet release reaction and also elicits a significantly higher epinephrine response in hypertensive men compared to normotensive controls.

whether smoking is a handicap for blood donation.17 In contrast numerous studies suggested that cigarette smoking increased platelet aggregation. A flow cytometric study established the presence of in vivo platelet activation immediately after smoking and a significant number of circu-lating platelets that are already in activated state in chronic

Platelets from smokers are also less responsive to nitric-oxide-mediated inhibition of aggregation than platelets from nonsmokers, 89 and the impaired release of platelet-derived nitric oxide in ...

Can stress cause high platelets? Yes, stress can indeed cause high platelets, a condition known as reactive thrombocytosis or secondary thrombocytosis. Disclaimer: This article is intended for informational purposes only and is specifically targeted towards medical students. It is not intended to be a substitute for informed professional ...

On the basis of their smoking characteristics, smokers were grouped as mild, moderate, and heavy. Result: Compared with nonsmokers, smokers showed significantly high values of MPV, and PDW (P < 0.05).